Showing posts with label Deficiency of Vitamin M and Vitamin B12. Show all posts
Showing posts with label Deficiency of Vitamin M and Vitamin B12. Show all posts

Deficiency of Vitamin M and Vitamin B12

Deficiency of Vitamin M and Vitamin B12
Deficiency of Vitamin M and Vitamin B12

Function of folate and vitamin B12 (cobalamin) are associated from the formation of red blood cells and in occurance of 1 of the essential the different parts of DNA (thymidine).

Insufficient pteroylglutamic acid and Vitamin B12

Pernicious anaemia (anemia on account of vitamin B12 deficiency) can be a condition in which vitamin B12 can not be absorbed by the stomach given it cannot produce intrinsic factor, which will join with vitamin B12 and transport it into your bloodstream.
Anemia may also be occurs because an excessive body's defence mechanism attacks the stomach cells that produce intrinsic factor (autoimmune reaction).

Other kinds of vitamin B12 deficiency may appear in:
# Vegetarian, because vitamin B12 is merely seen in animal products
# Patients with inherited disorders, or activities that hinder the transport in this vitamin.

Vitamin M deficiency can happen in expectant women whose intake of food containing a bit of greens and legumes, that includes a great deal of folate.
Babies can experience deficit of vitamin M when pteroylglutamic acid content in formula milk is low.

One lack of this vitamin causes serious anemia (pernicious anemia), the location where the red blood cells within a small amount but larger size.
Symptoms include pallor, weakness, decreased gastric acid secretion and nerve damage (neuropathy).
Neuropathy mainly occurs on account of insufficient vitamin B12.

Diagnosing vitamin B12 deficiency or folic acid is based on the finding of anemia with red blood cells which can be large plus the low levels found in the blood of one or both these vitamins.

Bone marrow examination showed immature precursors of red blood cells which have been large, that can confirm the diagnosis.

Treatment for pernicious anaemia for instance vitamin B12 injections every 4 weeks.

Treatment of pteroylglutamic acid deficiency will be the administration of folic acid orally (swallowed).


EXCESS ACID Folic

Vitamin M could potentially cause poisoning in a few circumstances.
At doses in excess of 100 times the recommended daily dose, could raise the frequency of seizures in patients with epilepsy and exacerbate neuronal damage in individuals who have problems with vitamin B12 deficiency.